鸚鵡熱衣原體免疫熒光玻片試劑盒

Chlamydophilia psittaci 4-spot Substrate Slide

廣州健侖生物科技有限公司

主要用途：用于檢測人/貓血清中的鸚鵡熱衣原體抗體

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鸚鵡熱衣原體免疫熒光玻片試劑盒

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【公司名稱】 廣州健侖生物科技有限公司
【】    楊永漢 
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但令人意外的是，圖西和倫德布拉德發(fā)現(xiàn)在端粒酶復(fù)合體充分組合后，它會迅速分解形成一個非活躍的“分解”復(fù)合體——事實(shí)上就是將開關(guān)調(diào)至“關(guān)閉”狀態(tài)。他們推測這種解體過程可以提供一種方式，讓端粒酶在細(xì)胞內(nèi)保持極低的濃度。盡管正常細(xì)胞內(nèi)不斷磨損的端粒酶會導(dǎo)致衰老，但癌癥細(xì)胞卻依賴提粒酶濃度，確保無節(jié)制的細(xì)胞增長。圖西和倫德布拉德發(fā)現(xiàn)的這個“關(guān)閉”開關(guān)可能有助于讓端粒酶的活躍度低于這一限度。
癌癥，雖然總是帶著危險的，但真正對生命造成威脅的是當(dāng)癌細(xì)胞開始擴(kuò)散到不同的領(lǐng)域和遍及全身時?，F(xiàn)在，美國密蘇里大學(xué)(MU)的研究人員發(fā)現(xiàn)，由細(xì)菌用作通訊系統(tǒng)的一個分子可以被操縱來阻止癌細(xì)胞擴(kuò)散。MU獸醫(yī)醫(yī)學(xué)院比較腫瘤學(xué)和表觀遺傳學(xué)實(shí)驗(yàn)室的助理研究教授和副主任Senthil Kumar說，這個通訊系統(tǒng)可用于“告訴”癌細(xì)胞如何行動，甚至是死亡的命令。
“在感染期間，細(xì)菌會釋放出分子，使得它們能夠彼此‘交談’，” Kumar說，他了這項(xiàng)研究。“所述信號根據(jù)釋放分子的類型，告訴其他細(xì)菌繁殖，逃避免疫系統(tǒng)甚至停止蔓延。我們發(fā)現(xiàn)，如果我們將‘停止蔓延’的細(xì)菌分子引入癌細(xì)胞中，這些細(xì)胞不但會停止擴(kuò)散，而且它們也將會開始死亡。”

Surprisingly, however, Tutsi and Lund Brad found that after the omerase complex has been fully assembled, it rapidly breaks down into an inactive "break down" complex - in effect switching the switch to " Disabled. They speculate that this disintegration process can provide a way for omerase to remain at very low concentrations in the cell. Although omerase, which is constantly abraded in normal cells, can cause aging, cancer cells rely on increasing omerase concentration to ensure uncontrolled cell growth. This "switch-off" switch, discovered by Tucci and Lund Brad, may help to keep omerase activity below this limit.
Cancer, while always dangerous, is a real threat to life as cancer cells begin to spread to different areas and spread throughout the body. Now researchers at the University of Missouri (MU) find that a molecule that bacteria use as a communication system can be manipulated to stop cancer cells from spreading. Senthil Kumar, professor and deputy director of assistant research at the MU Veterinary Medical School's Comparative Oncology and Epigenetics Laboratory, said the communication system can be used to "l" how cancerous cells act or even die.
"During infection, bacteria release molecules so they can talk to each other," Kumar said, who led the study. "The signal ls other bacteria to breed, escape the immune system, or even stop spreading, depending on the type of molecule released, and we found that if we introduce 'stop-going' bacterial molecules into cancer cells, they not only stop spreading, but they also It will start to die. "