甲型流感病毒H4亞型核酸檢測(cè)試劑盒

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甲型流感病毒H4亞型核酸檢測(cè)試劑盒

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【公司名稱】 廣州健侖生物科技有限公司
【市場(chǎng)部】    楊永漢

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【騰訊  】 2042552662
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-103室

皮膚和粘膜的顏色隨血流的顏色而變化。血液的紅色是由于紅細(xì)胞內(nèi)含有血紅蛋白。當(dāng)血紅蛋白充分地和氧結(jié)合，成為氧合血紅蛋白時(shí)，它的顏色是鮮紅的；當(dāng)它放出了氧，成為還原血紅蛋白時(shí)，顏色就變?yōu)榘导t。動(dòng)脈和毛細(xì)血管里的血，含氧合血紅蛋白多而還原血紅蛋白少，因此它的顏色鮮紅，透過(guò)薄的粘膜和半透明的指甲，紅色仍明顯。皮膚較厚，且含有色素，因而是白里透紅或微棕色透紅。靜脈血因含還原血紅蛋白多、氧合血紅蛋白少，所以它是暗紅色，透過(guò)皮膚，就呈現(xiàn)青紫色。手臂上一條一條的一般所稱的“青筋”就是靜脈。苯胺、硝基苯和亞硝酸鹽等化學(xué)品可使血紅蛋白變?yōu)樽冃匝t蛋白，這種血紅蛋白本身就是紫色的。因此，凡粘膜、指甲和皮膚里的毛細(xì)血管和小動(dòng)脈里血液的氧合血紅蛋白減少，而還原血紅蛋白增多或出現(xiàn)變性血紅蛋白的時(shí)候，都會(huì)出現(xiàn)紫紺。

病因

根據(jù)引起紫紺的病因不同，臨床上將紫紺分為兩大類(lèi)：血液中還原血紅蛋白增多引起的紫紺、血液中異常血紅蛋白衍生物引起的紫紺。其中，這兩類(lèi)當(dāng)中數(shù)前者zui為多見(jiàn)，也更具有臨床意義。下面我們?cè)敿?xì)進(jìn)行進(jìn)一步分類(lèi)。

血中含有還原血紅蛋白增加
根據(jù)不同病因引起又可以分為中央型紫紺、周?chē)妥辖C、混合型紫紺。

中央性紫紺
中央性紫紺（中心性發(fā)紺）由于心臟、呼吸系統(tǒng)疾病，導(dǎo)致血氧飽和度降低，臨床表現(xiàn)為彌漫性發(fā)紺。

心源性發(fā)紺：常見(jiàn)于先天性心臟病，如法洛四聯(lián)癥，因?yàn)閯?dòng)靜脈分流，靜脈血未經(jīng)肺泡即直接進(jìn)入動(dòng)脈系統(tǒng)。如分流量占心臟輸出量30%以上，則發(fā)生發(fā)紺。
肺源性發(fā)紺：見(jiàn)于肺動(dòng)脈受損，因?yàn)榉闻萃獠蛔?、肺通氣與肺血流比例失調(diào)、肺泡膜氧彌散功能障礙等因素所致。常見(jiàn)的疾病有阻塞性肺氣腫、肺炎、肺淤血、肺水腫等。
周?chē)妥辖C
周?chē)妥辖C（周?chē)园l(fā)紺）由于周?chē)h(huán)障礙所致。其臨床表現(xiàn)特點(diǎn)是發(fā)紺常出現(xiàn)于肢體的末端，比如耳垂、顏面等。

靜脈淤血：比如下肢靜脈栓塞、靜脈曲張；
心排血量減少：嚴(yán)重休克時(shí)，因?yàn)橹車(chē)苎骶徛把苁湛s，導(dǎo)致組織缺血缺氧。
肢體末端循環(huán)障礙：如雷諾病、雷諾現(xiàn)象。
周?chē)鷦?dòng)脈阻塞：動(dòng)脈硬化、閉塞性脈管炎等。

The color of the skin and mucous membranes changes with the color of the bloodstream. Red blood is due to red blood cells contain hemoglobin. When hemoglobin is fully bound to oxygen and becomes oxy-hemoglobin, its color is bright red; when it releases oxygen and becomes hemoglobin, the color turns dark red. Arteries and capillaries in the blood, oxygenated hemoglobin reduction of hemoglobin and less, so its bright red color, through the thin mucous membrane and translucent nails, the red is still obvious. Thicker skin, and contains pigment, which is white or reddish brown. Venous blood contains more hemoglobin due to reduction, oxygenated hemoglobin less, so it is dark red, through the skin, it appears blue. The generally known "veins" on the arms are veins. Aniline, nitrobenzene and nitrite and other chemicals can make hemoglobin into denatured hemoglobin, the hemoglobin itself is purple. Therefore, where mucosa, nails and skin in the capillaries and arterioles in the blood oxygenated hemoglobin decreased, and the reduction of hemoglobin increased or denatured hemoglobin when there will be cyanosis.

Etiology

According to the different causes of cyanosis, cyanosis is clinically divided into two categories: cyanosis caused by increased hemoglobin in the blood, and cyanosis caused by abnormal hemoglobin derivatives in the blood. Among them, the former two types of the former is the most common, but also more clinically significant. Below we carry out further classification.

Blood contains reduced hemoglobin increased
According to different causes can be divided into central cyanosis, peripheral cyanosis, mixed cyanosis.

Central cyanosis
Central cyanosis (central cyanosis) due to the heart, respiratory diseases, leading to decreased oxygen saturation, the clinical manifestations of diffuse cyanosis.

Cardiac cyanosis: Common in congenital heart disease, such as tetralogy of Fallot, because arteriovenous shunt, venous blood directly into the arterial system without alveolar. If the sub-flow accounted for more than 30% of cardiac output, the occurrence of cyanosis.
Lung-derived cyanosis: seen in the pulmonary artery damage due to inadequate alveolar ventilation, pulmonary ventilation and pulmonary blood flow imbalance, alveolar oxygen diffusion dysfunction and other factors. Common diseases are obstructive emphysema, pneumonia, pulmonary congestion, pulmonary edema and so on.
The surrounding cyanosis
Peripheral cyanosis (peripheral cyanosis) due to the surrounding circulatory disorders. The clinical manifestations of cyanosis often appears in the extremities, such as earlobe, face and so on.

Venous congestion: such as lower extremity venous thrombosis, varicose veins;
Cardiac output reduced: severe shock, because of slow peripheral blood flow and vasoconstriction, leading to tissue ischemia and hypoxia.
Circumferential limbs disorders: such as Raynaud's disease, Raynaud's phenomenon.
Peripheral artery obstruction: atherosclerosis, occlusive vasculitis and so on.